Preliminary experiments point out conolidine may perhaps inhibit distinct ion channels, lowering neuronal excitability and restricting ache signals. This mechanism is particularly suitable in neuropathic agony, in which irregular signaling causes persistent distress. Furthermore, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to suffering perception. ... https://joycee046omh7.slypage.com/profile