Preliminary experiments show conolidine may well inhibit specific ion channels, lowering neuronal excitability and restricting agony signals. This mechanism is particularly appropriate in neuropathic discomfort, wherever irregular signaling results in persistent pain. Moreover, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to pain notion. Computational... https://carlm135hyr7.wikilowdown.com/user