Mitochondrial dysfunction and involved enhanced ROS are associated with aging, and specifically to senescence and inflammation. The maintenance of mitochondrial action has actually been a primary therapeutic goal as murine experiments have revealed valuable effects of PGC1α overexpression on many aging-relevant biological processes including metabolism and ROS control(Dillon et al. https://cristianvwtok.blogaritma.com/28001773/what-does-telomer-bölge-nedir-mean